[Retracted] miR-29c Inhibits Renal Interstitial Fibrotic Proliferative Properties through PI3K-AKT Pathway

Figure 3

miR-29c activated the PI3K-AKT pathway in NRK-52E cultures. (a) The negative control and overexpressing miR-29c cultures were exposed to TGF-β1, and then, the total protein and phosphorylation of PI3K/Akt expression profiles were evaluated through WB. (b) Negative control and overexpressed miR-29c cultures were exposed to TGF-β1, with PI3K relative expression subsequently evaluated through RT-qPCR. (c) Negative control and overexpressing miR-29ccultures were exposed to TGF-β1, with PI3K relative expression subsequently evaluated through RT-qPCR. (d) The negative control and overexpressing miR-29ccultures were exposed to TGF-β1, with AKT relative expression subsequently evaluated through RT-qPCR. (e) The negative control and overexpressing miR-29ccultures were exposed to TGF-β1, and then, AKT phosphorylation relative expression was subsequently evaluated through RT-qPCR. and .

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