Research Article

The Role of Overexpressed Apolipoprotein AV in Insulin-Resistant Hepatocytes

Figure 1

Insulin-resistant HepG2 cell model construction. (a) Glucose uptake rate and (b) liver glycogen synthesis after HepG2 cells treated with 5 × 10−5, 5 × 10−6, 5 × 10−7, and 5 × 10–8 mol/L bovine insulin. (c) APOAV protein expression in insulin-resistant HepG2 cells.
(a)
(b)
(c)