[Retracted] Establishment and Verification of a Gene Signature for Diagnosing Type 2 Diabetics by WGCNA, LASSO Analysis, and In Vitro Experiments

Figure 9

Silencing BST2 ameliorates apoptosis of glucotoxicity islet β cell models and influences the activation of TGF-β and P53 pathways. (a) RT-qPCR of the expression of BST2 mRNA in islet β cells with si-NC or si-BST2 transfection. (b) and (c) Flow cytometry of apoptotic levels of glucotoxicity models and normal islet β cells. (d) and (e) Flow cytometry of apoptotic levels of glucotoxicity models under si-NC or si-BST2 transfection. (f)–(h) Western blot of the expression of TGF-β and P53 proteins in glucotoxicity models and normal islet β cells. (i)–(k) Western blot of the expression of TGF-β and P53 proteins in glucotoxicity models under si-NC or si-BST2 transfection. ; .

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