Analysis of Therapeutic Targets of A Novel Peptide Athycaltide-1 in the Treatment of Isoproterenol-Induced Pathological Myocardial Hypertrophy

Figure 3

ATH-1 decreased the protein expression of Hspa1, p-ERK, p-JNK, and p-p38 in hypertrophic myocardium and enhanced the activity of T-SOD. (a) Differentially abundant proteins identified in the iTRAQ experiment. (b) The protein expression of Hspa1 (). and . (c) The activity of T-SOD in each group (). and . (d) The protein expression of ERK1/2 and p-ERK1/2 (). ns indicates none significance, , , and . (e) The protein expression of JNK and p-JNK (). ns indicates none significance, . (f) The protein expression of p38 MAPK and p-p38 MAPK (). ns indicates none significance, and .

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