[Retracted] Hypoxia-Induced Nestin Regulates Viability and Metabolism of Lung Cancer by Targeting Transcriptional Factor Nrf2, STAT3, and SOX2

Table 1

Relationship of the Nestin expression in hNSCLC with clinical and pathological factors.

	Patients	NESTIN-low, n (%)	NESTIN-high, n (%)	(value)
Sex
Female	25	17 (54.0)	8 (46.0)	0.548
Male	33	18 (43.9)	15 (56.1)
Age (y)
≤60	36	20 (44.7)	16 (55.3)	0.298
>60	22	12 (58.7)	10 (41.3)
Smoking
Yes	34	28 (60.7)	6 (39.3)	0.155
No	24	14 (33.3)	10 (66.7)
Diffrentitian
Good	23	15 (67.3)	8 (32.7)	0.047
Moderate	17	8 (34.5)	9 (63.5)
Poor	18	12 (67.4)	4 (32.6)
TNM stage
I	33	15 (61.7)	18 (38.3)	0.189
II	12	7 (56.0)	5 (43.0)
III	13	6 (45.9)	7 (56.1)
Histology
Adenocarcinoma	29	19 (53.8)	10 (46.2)	0.003
Squamous cell carcinoma	26	13 (50.3)	13 (49.7)
Large cell carinoma	3	1 (50.0)	2 (50.0)
Lymph node metastasis
N0	23	14 (72.2)	9 (27.8)	0.009
N1	20	9 (61.3)	11 (38.7)
N2	15	10 (78.9)	5 (21.1)
MVD expression
High	30	19 (64.9)	11 (35.1)	0.22
Low	22	14 (56.9)	8 (43.1)
Lvd expresssion
High	34	20 (48.3)	14 (51.7)	0.874
Low	24	13 (73.6)	9 (26.4)
VEGF expression
High	29	15 (61.9)	14 (38.1)	0.754
Low	29	18 (40.0)	11 (60.0)
COX-2 expression
High	28	17 (68.3)	11 (31.7)	0.269
Low	30	22 (62.0)	8 (38.0)