[Retracted] Electroacupuncture Treats Myocardial Infarction by Influencing the Regulation of Substance P in the Neurovascular to Modulate PGI2/TXA2 Metabolic Homeostasis via PI3K/AKT Pathway: A Bioinformatics-Based Multiomics and Experimental Study

Figure 3

SP affects the PGI2/TXA2 balance by influencing the PI3K/AKT pathway. (a) Determination of PGI2 concentrations in mouse serum by ELISA; (b) determination of TXA2 concentrations in mouse serum by ELISA; (c) RT-PCR of Myd88, Irak1, Tab1, Nf-κb1, Pik3c1, Akt, and Mtor gene expression in infarcted myocardial tissues of mice; (d) expression of MTOR, p-AKT, AKT, and GAPDH genes in infarcted myocardial tissues of mice by western blot; (e) expression of Myd88, Irak1, Tab1, Nf-κb1, Pik3c1, Akt, and Mtor genes after SP treatment; (f) expression of MTOR, p-AKT, AKT, and MTOR after SP treatment by western blot.

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