Reduced Circulating Endothelial Progenitor Cells and Downregulated GTCPH I Pathway Related to Endothelial Dysfunction in Premenopausal Women with Isolated Impaired Glucose Tolerance

<div>The correlations between circulating EPCs or NO production and FMD. The number of circulating EPCs evaluated by flow cytometric analysis (a) or by cell culture (b) strongly correlated with FMD. The migratory (c) or proliferate (d) activity of circulating EPCs also correlated with FMD. Both plasma NO level (e) and NO secretion by EPCs (f) correlated with FMD. LDL, low density lipoprotein; CD, cluster of differentiation; KDR, kinase-insert domain receptor; FMD, flow-mediated brachial artery dilatation; EPCs, endothelial progenitor cells; NO, nitric oxide.</div>

Cardiology Research and Practice

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Figure 3: Reduced Circulating Endothelial Progenitor Cells and Downregulated GTCPH I Pathway Related to Endothelial Dysfunction in Premenopausal Women with Isolated Impaired Glucose Tolerance 

Figure 3 | Reduced Circulating Endothelial Progenitor Cells and Downregulated GTCPH I Pathway Related to Endothelial Dysfunction in Premenopausal Women with Isolated Impaired Glucose Tolerance 