Integrative Bioinformatics Analysis Reveals That Infarct-Mediated Overexpression of Potential miR-662/CREB1 Pathway-Induced Neuropeptide VIP Is Associated with the Risk of Atrial Fibrillation: A Correlation Analysis between Myocardial Electrophysiology and Neuroendocrine

<div>GSEA of CREB1 and VIP in AF: (a) pathways in AF in which the CREB1 high-expression group is enriched in the KEGG gene set; (b) pathways in AF in which the VIP high-expression group is enriched in the KEGG gene set. Curve peaks above the horizontal axis indicate that the pathway is upregulated in AF; otherwise, it represents downregulation.</div>

Disease Markers

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Figure 5

Figure 5: Integrative Bioinformatics Analysis Reveals That Infarct-Mediated Overexpression of Potential miR-662/CREB1 Pathway-Induced Neuropeptide VIP Is Associated with the Risk of Atrial Fibrillation: A Correlation Analysis between Myocardial Electrophysiology and Neuroendocrine 

Figure 5 | Integrative Bioinformatics Analysis Reveals That Infarct-Mediated Overexpression of Potential miR-662/CREB1 Pathway-Induced Neuropeptide VIP Is Associated with the Risk of Atrial Fibrillation: A Correlation Analysis between Myocardial Electrophysiology and Neuroendocrine 