Inhibition of Helicobacter pylori CagA-Induced Pathogenesis by Methylantcinate B from Antrodia camphorata
Figure 5
MAB attenuates H. pylori-induced IL-8 activity. AGS cells were infected with H. pylori in the absence or presence of MAB for 6 h. Cell lysates and culture supernatants were prepared for IL-8 luciferase activities (a) and IL-8 secretions (b), respectively. ** was considered as statistically significant. MAB−, not treated with MAB; MAB+, treated with MAB.
