[Retracted] LINC00035 Transcriptional Regulation of SLC16A3 via CEBPB Affects Glycolysis and Cell Apoptosis in Ovarian Cancer

Figure 6

A schematic diagram showing how the LINC00035/CEBPB/SLC16A3 pathway regulates the development of OC. LINC00035 was triggered in OC and could recruit the transcription factor CEBPB into the SLC16A3 promoter region, thus increasing the SLC16A3 transcription. Abundance of SLC16A3 inhibited OC cell viability, migration, invasion, and glycolysis and induced the apoptosis.