Huangjia Ruangan Granule Inhibits Inflammation in a Rat Model with Liver Fibrosis by Regulating TNF/MAPK and NF-κB Signaling Pathways

Figure 8

Schematic diagram of the proposed mechanisms underlying the protective effects of HJRG inhibiting CCl₄-induced liver fibrosis in rats. The mechanism of HJRG may be inhibiting the TNFR1-mediated MAPK and NF-κB signaling pathways, reducing the inflammatory response, and ameliorating oxidative stress, thereby inhibiting liver fibrosis.