[Retracted] Role and Mechanism of Epithelial-Mesenchymal Transition Mediated by Inflammatory Stress-Induced TGF-<i>β</i>1 in Promoting Arteriovenous Fistula Stenosis

<div>Comparison of <i>α</i>-SMA and CD31/vWF expressions in both Groups. (a) The expression of fluorescein antibody SMA in smooth muscle cells of SMA and CD32/vWF was not found; (b) the expression of fluorescein antibody SMA in smooth muscle cells; (c) the expression of cytofluorescein antibody SMA in proliferative myofibroblasts; (d) the expression of fluorescein antibody SMA in smooth muscle cells and proliferative myofibroblasts; (e) the expression of fluorescein antibody CD31/vWF in endothelial cells; (f) the expression of fluorescein antibody -SMA and vWF in endothelial cells.</div>

Evidence-Based Complementary and Alternative Medicine

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Figure 3

Figure 3: [Retracted] Role and Mechanism of Epithelial-Mesenchymal Transition Mediated by Inflammatory Stress-Induced TGF-<i>β</i>1 in Promoting Arteriovenous Fistula Stenosis 

Figure 3 | [Retracted] Role and Mechanism of Epithelial-Mesenchymal Transition Mediated by Inflammatory Stress-Induced TGF-β1 in Promoting Arteriovenous Fistula Stenosis 