Urinary Sodium Concentration Is an Independent Predictor of All-Cause and Cardiovascular Mortality in a Type 2 Diabetes Cohort Population

Table 6

Risk of cardiovascular death according to Cox model left and Fine and Gray competing risk right model.

	Adjusted; hazard ratio; (95% CI)	value	Adjusted; subhazard ratio⁢; (95% CI)	value
Age	1.76 (1.50 to 2.07)	<0.001	1.53 (1.30 to 1.79)	<0.001
Sex: (reference men)	0.66 (0.50 to 0.86)	0.002	0.73 (0.56 to 0.95)	0.02
BMI	1.19 (1.04 to 1.36)	0.01	1.13 (0.99 to 1.28)	0.07
Use of Insulin	1.62 (1.20 to 2.19)	0.002	1.57 (1.15 to 2.14)	0.005
uACR	1.26 (1.11 to 1.42)	<0.001	1.19 (1.04 to 1.36)	0.01
	0.76 (0.66 to 0.88)	<0.001	0.82 (0.71 to 0.95)	0.007
NT-proBNP	1.90 (1.66 to 2.18)	<0.001	1.80 (1.56 to 2.08)	<0.001

Hazard ratios were standardized by calculating them for 1-SD increment of each continuous variable. History of uACR, urinary albumin to creatinine concentration ratio; , urinary sodium concentration; NT-proBNP, N-terminal pro-brain natriuretic peptide.
uACR and NT-proBNP are log-transformed.
Cox proportional subhazards ratios for the risk of cardiovascular death are computed according to Fine and Gray competing risk models when taking into account the competing risk of noncardiovascular death.