KLF4 Promotes Diabetic Chronic Wound Healing by Suppressing Th17 Cell Differentiation in an MDSC-Dependent Manner

Figure 2

Kruppel-like factor 4 (KLF4) activation by APTO-253 accelerated pressure ulcer (PU) in ob/ob mice accompanied by increased CD11b⁺Gr-1⁺ myeloid-derived suppressor cells (MDSCs) and decreased Th17 cells.

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