Beta-Hydroxybutyric Acid Inhibits Renal Tubular Reabsorption via the AKT/DAB2/Megalin Signalling Pathway

Figure 2

Percentages of patients with increased glomerular and tubular injury in DK patients. ACR, albumin creatinine ratio; uβ2-MG, urine β2-microglobulin; uGAL, urine β-galactosidase; uIgG, urine Immunoglobulin G; uTF, urine Transferrin; uNAG, urine N-acetyl-β-glucosaminidase; uRBP, urine retinol-binding protein; NC, normal control; NDK, non-diabetic ketosis; DK, diabetic ketosis. ACR≥30mg/g was defined as between NC,NDK, and DK groups, between before and after ketosis control; urine β2-MG≥0.3mg/l was defined as , between NC, NDK, and DK groups, , compared before and after ketosis control; urine GAL≥15U/l was recorded as , between NC, NDK, and DK groups, between before and after ketosis control; urine IgG≥17.5mg/l was recorded as between NC,NDK and DK groups, between before and after ketosis control; urine NAG≥12U/l was recorded as between NC,NDK and DK groups, between before and after ketosis control; urine RBP≥0.7mg/l was recorded as between NC, NDK, and DK groups, between before and after ketosis control; urine TF≥5mg/l was recorded as between NC,NDK and DK groups, between before and after ketosis control.

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