Helicobacter pylori and Epstein–Barr Virus Infection in Gastric Diseases: Correlation with IL-10 and IL1RN Polymorphism
Table 2
Gene analysis of our sample data.
Parameters
Sample data
Polymorphism of gene vacA
s1m1-m2i1
2.27% (1/44)
s1m2i2
4.55% (2/44)
s1m2i1
6.82% (3/44)
s1m1i1
40.91% (18/44)
s2m2i2
45.45% (20/44)
Gene cagA ()
Negative
47.73% (21/44)
Positive
52.27% (23/44)
Gene oipA ()
“OFF”
47.73% (21/44)
“ON”
52.27% (23/44)
Polymorphism IL-1RN
1/1
56.35% (54/96)
1/2
21.88% (21/96)
2/2
18.75 (18/96)
1/3
2.08% (2/96)
4/4
1.04% (1/96)
Polymorphism IL-10 (−1082)
AA
47.92% (46/96)
AG
29.17% (28/96)
GG
22.92% (22/96)
H. pylori resistant
27.27% (12/44)
H. pylori sensible
72.73% (32/44)
HP = H. pylori; GCNA = inactive chronic gastritis; GCA = active chronic gastritis; KG = gastric cancer; ML = MALT lymphoma; cases where gene vacA was present.
