Deficiency of kin17 Facilitates Apoptosis of Cervical Cancer Cells by Modulating Caspase 3, PARP, and Bcl-2 Family Proteins

<div>The potential mechanism diagram of kin17 knockdown involved in cervical cancer cell apoptosis. PARP, poly ADP-ribose polymerase; CL-PARP, cleaved poly ADP-ribose polymerase; Bim, Bcl-2 interacting mediator of cell death; Bcl-xL, B-cell lymphoma-xL; Bad, Bcl2 associated death promoter.</div>

Journal of Oncology

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Figure 4

Figure 4: Deficiency of kin17 Facilitates Apoptosis of Cervical Cancer Cells by Modulating Caspase 3, PARP, and Bcl-2 Family Proteins 