Uroprotective and Hepatoprotective Potential of Anagallis arvensis against the Experimental Animal Model

Table 3

Effect of A. arvensis on nitric oxide, catalase enzyme and glutathione peroxide.

Groups	Control (Mean ± SEM)	Diseased control (Mean ± SEM)	Standard drug-treated (Mean ± SEM)	MEOAA 250 mg/kg treated (Mean ± SEM)	MEOAA 500 mg/kg treated (Mean ± SEM)
Nitric oxide (U/mg)	19.00 ± 0.577	50.66 ± 1.202	27.66 ± 1.453	30.33 ± 0.33	26.66 ± 0.88
Catalase (U/mg)	23.33 ± 0.66	6.33 ± 1.76	19.66 ± 0.88	18.00 ± 0.55	21.00 ± 0.57
Glutathione peroxide (U/mg)	26.00 ± 0.57	8.66 ± 0.33	23.33 ± 0.88	20.00 ± 0.55	23.00 ± 0.57
IL-6 (pg/ml)	30.75 ± 1.49	110.5 ± 1.04	83 ± 0.04	73 ± 0.91	64.5 ± 0.64
TNF-α (pg/ml)	16.25 ± 1.11	47.75 ± 0.75	39.5 ± 0.86	36 ± 0.41	30.75 ± 0.85

Determination of nitric oxide, catalase enzyme, glutathione peroxide, IL-6, and TNF-α levels in the blood for control, diseased control (cyclophosphamide 150 mg/kg), standard drug (Mesna 40 mg/kg)-treated and methanol extract of A. arvensis (MEOAA) (250 mg/kg and 500 mg/kg)-treated group of rats. ( compared to the control group, compared to diseased control group; n = 4) (IL-6; interleukin-6, TNF-α; tumor necrosis factor-α).