Astragaloside IV Inhibits Oxidative Stress-Induced Mitochondrial Permeability Transition Pore Opening by Inactivating GSK-3 via Nitric Oxide in H9c2 Cardiac Cells
Figure 1
Western blotting analysis of GSK-3β phosphorylation at Ser9 and total GSK-3β protein in cardiac H9c2 cells. Astragaloside IV (20~100 μM) increased GSK-3β phosphorylation in a dose-dependent manner. Data are mean ± SD for 6 independent experiments performed in duplicate. * compared to control group (t-test).