Overexpression of the Aryl Hydrocarbon Receptor (Ahr) Mediates an Oxidative Stress Response following Injection of Fine Particulate Matter in the Temporal Cortex

<div>Immunofluorescence examination of AhR expressions. The levels of the AhR protein (red) in the ipsilateral primary temporal cortex were increased in the PM<sub>2.5</sub> group compared to the saline group (blue: DAPI).</div>

Oxidative Medicine and Cellular Longevity

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Figure 4

Figure 4: Overexpression of the Aryl Hydrocarbon Receptor (Ahr) Mediates an Oxidative Stress Response following Injection of Fine Particulate Matter in the Temporal Cortex 

Figure 4 | Overexpression of the Aryl Hydrocarbon Receptor (Ahr) Mediates an Oxidative Stress Response following Injection of Fine Particulate Matter in the Temporal Cortex 