[Retracted] Breast Cancer Exosome-Derived miR-425-5p Induces Cancer-Associated Fibroblast-Like Properties in Human Mammary Fibroblasts by TGF<i>β</i>1/ROS Signaling Pathway

<div>The schematic diagram depicts the regulatory mechanism of exosomes deliver miR-425-5p in HMF switch to CAF phenotype. Exosomal miR-425-5p promoted HMF switch to CAF phenotype via inhibiting TGF<i>β</i>RII expression to activate TGF<i>β</i>1 signaling pathway, thereby playing tumor-promoting abilities in normal fibroblasts.</div>

Oxidative Medicine and Cellular Longevity

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Figure 10

Figure 10: [Retracted] Breast Cancer Exosome-Derived miR-425-5p Induces Cancer-Associated Fibroblast-Like Properties in Human Mammary Fibroblasts by TGF<i>β</i>1/ROS Signaling Pathway 

Figure 10 | [Retracted] Breast Cancer Exosome-Derived miR-425-5p Induces Cancer-Associated Fibroblast-Like Properties in Human Mammary Fibroblasts by TGFβ1/ROS Signaling Pathway 