[Retracted] Macrophage-Derived Exosomes in TLR9<sup>-/-</sup> Mice Ameliorate Sepsis-Induced Mitochondrial Oxidative Stress and Apoptosis in Cardiomyocytes

<div>Myeloid deficiency of TLR9 improved morphology of cardiomyocytes in septic mice. (a) WT and TLR9<sup>-/-</sup> BMT mice in the control group showed health characteristics of cardiac cell structure. (b) TLR9<sup>-/-</sup> BMT mice improved or inhibited the extensive cellular shrinkage, chromatin condensation, and nuclear fragmentation compared with WT BMT mouse heart. Scar bar: 100 <i>μ</i>m.</div>

Oxidative Medicine and Cellular Longevity

fig1

Figure 1

Figure 1: [Retracted] Macrophage-Derived Exosomes in TLR9<sup>-/-</sup> Mice Ameliorate Sepsis-Induced Mitochondrial Oxidative Stress and Apoptosis in Cardiomyocytes 

Figure 1 | [Retracted] Macrophage-Derived Exosomes in TLR9-/- Mice Ameliorate Sepsis-Induced Mitochondrial Oxidative Stress and Apoptosis in Cardiomyocytes 