The Polysaccharides from the Aerial Parts of <i>Bupleurum chinens</i>e DC Attenuate Epilepsy-Like Behavior through Oxidative Stress Signaling Pathways

<div>The regulation of oxidative stress pathways of KA-induced rats with treatment. (a) Western blotting in KA-induced epilepsy rats with treatment; (b) the expression of TREM 2 in KA-induced epilepsy rats with treatment; (c) the expression of PI3K in KA-induced epilepsy rats with treatment; (d) the expression of AKT in KA-induced epilepsy rats with treatment; (e) the expression of P-AKT in KA-induced epilepsy rats with treatment; (f) the expression of GSK-3<i>β</i> in KA-induced epilepsy rats with treatment.</div>

Oxidative Medicine and Cellular Longevity

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Figure 9

Figure 9: The Polysaccharides from the Aerial Parts of <i>Bupleurum chinens</i>e DC Attenuate Epilepsy-Like Behavior through Oxidative Stress Signaling Pathways 

Figure 9 | The Polysaccharides from the Aerial Parts of Bupleurum chinense DC Attenuate Epilepsy-Like Behavior through Oxidative Stress Signaling Pathways 