Rosiglitazone Restores Endothelial Dysfunction in a Rat Model of Metabolic Syndrome through PPARγ- and PPARδ-Dependent Phosphorylation of Akt and eNOS

Figure 1

Biometric characteristics of rats on normal chow diet (Chow, 𝑛=10) and high-fat diet (HFD, 𝑛=12) for 32 weeks. (a) Body weight; (b) Visceral fat percentage; (c) Fasting plasma insulin level; (d) Fasting plasma glucose; (e) Fasting plasma triglycerides; (f) Systolic blood pressure by tail-cuff method. Data are means ± SEM *𝑃<0.05, **𝑃<0.01 compared to Chow.

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