Increased Death of Peripheral Blood Mononuclear Cells after TLR4 Inhibition in Sepsis Is Not via TNF/TNF Receptor-Mediated Apoptotic Pathway

<div>The bar charts with one standard error show caspase-3, caspase-8, and caspase-9 activities in cultured PBMCs with/without LPS, rhHMGB1, and anti-TLR4 antibody treatment (PBMCs = peripheral blood mononuclear cells; LPS = lipopolysaccharide; rhHMGB1 = recombinant human high-mobility group box 1; TLR = toll-like receptor).</div>

Mediators of Inflammation

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Figure 3

Figure 3: Increased Death of Peripheral Blood Mononuclear Cells after TLR4 Inhibition in Sepsis Is Not via TNF/TNF Receptor-Mediated Apoptotic Pathway 

Figure 3 | Increased Death of Peripheral Blood Mononuclear Cells after TLR4 Inhibition in Sepsis Is Not via TNF/TNF Receptor-Mediated Apoptotic Pathway 